C Treatments How Work

C Treatments work by blocking the action of the enzyme PDE-5 (phosphodiesterase-5), which is involved in the regulation of blood flow to the penis. By blocking PDE-5, cGMP (cyclic guanosine monophosphate) levels are increased, which relax the smooth muscles in the blood vessels of the penis and increase blood flow, resulting in an erection.

Here's a step-by-step explanation of how C treatments work:

Sexual stimulation: During sexual arousal, the release of nitric oxide (NO) is triggered in the penile tissues.

Activation of guanylate cyclase: NO activates the enzyme guanylate cyclase, which converts guanosine triphosphate (GTP) to cGMP.

Increased cGMP levels: cGMP acts as a signaling molecule that relaxes the smooth muscles in the blood vessels of the penis. This relaxation allows more blood to flow into the penis, causing an erection.

PDE-5 inhibition: C treatments work by inhibiting the enzyme PDE-5, which breaks down cGMP. By blocking PDE-5, cGMP levels are increased and sustained, leading to a longer-lasting erection.

Erection achieved: With increased blood flow and sustained cGMP levels, the penis becomes erect and remains firm during sexual activity.

Metabolism and elimination: After ejaculation, the levels of cGMP decrease as PDE-5 breaks down the remaining cGMP. The C is then metabolized and eliminated from the body through urine and feces.

It's important to note that C treatments are only effective in the presence of sexual stimulation. They do not directly induce an erection but rather enhance the natural erectile response to sexual arousal by facilitating increased blood flow to the penis.